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Unytii - Suppléments et Produits Naturels
MethylPlus
Ships from Quebec💰
Description
- Methylation agents and cofactors
- Lowers dangerous homocysteine levels
- Reduces the risk of cardiovascular disease
- Prevents blood vessel damage
- Slows the aging process
Methylation is a key process essential for the proper functioning of all cells in the body and helps prevent aging and disease. Methylation lowers homocysteine, a toxic amino acid that significantly increases the risk of heart disease, cancer, bone loss, joint degeneration, mood imbalance, and cognitive decline.
MethylPlus contains all the required cofactors and methyl donors for methylation processes and for the conversion of homocysteine into useful amino acids: vitamin B6, vitamin B12, folate, and trimethylglycine (TMG). This combination of nutrients has been shown to significantly reduce homocysteine levels after only 6 weeks of supplementation. TMG alone can reduce plasma homocysteine levels by up to 30%. Furthermore, vitamins B, particularly B6, B12, and folate, have been shown to reduce the risk of stroke due to their ability to lower homocysteine.
It has recently been discovered that a large portion of the population has an MTHFR gene mutation that prevents them from converting folate into its usable form, L-5-MTHF. This leads to folate deficiency and decreased methylation, resulting in high homocysteine levels. The folate used in MethylPlus is in the L-5-MTHF form, making it an ideal supplement for individuals with the MTHFR mutation.
The vast majority of people do not obtain the specific nutrients that promote methylation from their diet. MethylPlus is an excellent supplement for general aging, for individuals with high homocysteine levels, and for those seeking support against age-related degenerative diseases of the bones, joints, nerves, and cognitive functions.
AOR's MethylPlus formula combines the maximum cofactors and methyl donors necessary for the conversion of homocysteine into useful amino acids, thereby helping to reduce the risk of heart disease.
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